re-cwd tse prion disease and cjd in humans ?
I will have to dig through the academic papers I have on Creutzfeldt-Jakob to post links for. I am still in the middle of a move and a ton of research, books and such are in boxes. These papers are not just from the US, but Europe as well.
Greetings katqanna, Hunters et al @
http://onyourownadventures.com/hunttalk/ .
My name is Terry S. Singeltary Sr., and I wish to submit this data on Chronic Wasting Disease CWD in Cervids, and risk factors there from.
AS a layperson, I have wasted 15 years daily (it seems at times), going from state to state, country to country, warning of this dreaded disease. not to many folks listened.
It’s mostly a political disease, spread by political and industry greed.
I lost my mother to the Heidenhain Variant of the Creutzfeldt Jakob Disease hvCJD, a rare subtype of the sporadic CJDs in humans, and these phenotypes are mounting, and they are of unknown origin, and NOT simply a happenstance of bad luck, or a funked out twisted protein that just happen to make this twist on it’s own, this in 85%+ of all human TSE disease i.e. sCJD, thus, I will never believe this hypothesis. in other words, the UKBSEnvCJD only theory was trash.
I came across this thread, and thought I must respond.
please use this science and information as you wish. I simply wanted to share, the rest of the story here about the cwd tse prion disease.
the shooting pen livestock industry will not like what I have to say, and what science I present.
Chronic Wasting Disease CWD, and other TSE prion disease, these TSE prions know no borders.
these TSE prions know no age restrictions.
The TSE prion disease survives ashing to 600 degrees celsius, that’s around 1112 degrees farenheit.
you cannot cook the TSE prion disease out of meat.
you can take the ash and mix it with saline and inject that ash into a mouse, and the mouse will go down with TSE.
Prion Infected Meat-and-Bone Meal Is Still Infectious after Biodiesel Production as well.
the TSE prion agent also survives Simulated Wastewater Treatment Processes.
IN fact, you should also know that the TSE Prion agent will survive in the environment for years, if not decades.
you can bury it and it will not go away.
The TSE agent is capable of infected your water table i.e. Detection of protease-resistant cervid prion protein in water from a CWD-endemic area.
it’s not your ordinary pathogen you can just cook it out and be done with. that’s what’s so worrisome about Iatrogenic mode of transmission, a simple autoclave will not kill this TSE prion agent.
I go from state to state trying to warn of the CWD and other TSE prion disease in other species, I just made a promise to mom. back then, there was no information.
so, I submit this to you all in good faith, and hope that you take the time to read my research of the _sound_, peer review science, not the junk science that goes with the politics $$$
right or left or teaparty or independent, you cannot escape the TSE prion disease.
there is a lot of science here to digest, but better digesting this _sound_ science, instead of the junk political science you will hear from the shooting pen industry.
I don’t care what you eat, whom you eat, or what party you are affiliated with, my problem is, when you consume these TSE prions, and then go enter the medical, surgical, dental, blood and tissue arena, then you risk exposing _me or MY_ family to the TSE prion disease via friendly fire, the pass it forward mode of transmission mission, or what they call iatrogenic CJD. all iatrogenic CJD is, is sporadic CJD, until the route and source of the TSE prion agent is proven.
I am NOT anti-hunter, I am or was a hunter (disabled with neck injury and other medical problems), I am a meat eater.
I just don’t care for stupid, and sometimes you just can’t fix stupid, Lord knows I have tried.
I do NOT advertise on these blogs, they are there for educational use. please read the science, maybe read it again, try and understand it, most is not rocket science, and then you have to make your own decisions, but it’s just good when your making these decisions about CWD, policy making there from, that you have all the science, and not just part of it.
I have much to say, there is over 15 years of daily research of the science, updated, transmission studies, FOIA requests, confidential documents, dockets submissions, and more on the CWD, TSE, prion disease. so instead of posting a million pages of data on this, I have put this science in different blogs. please sort by topics of your concern, and then _please_ read the source data, where these studies were done, and what they consisted of. I do not pretend to know everything, and I am not here pretending this, I simply am here to try and educate, with what I have learned over the past 15+ years of daily research and debate on this topic of CWD, TSE prion, aka mad cow type disease.
now the shooting pen owners, the sperm mills, antler mills, captive livestock breeders, they all will be up in arms over this post, then you will have others say that I am a scare monger, a tree hugger, a vegan, anti this and that. I am none of these. you will ultimately have to make your own minds up. ...
Good Luck !
we will start here, my submission to the state of Missouri recently ;
My submission as follows ;
snip...
I guess we will start with the question, how much money can one state afford for one CWD infected game farm, and the ramifications there from ?
I will list one example here ;
how many states have $465,000., and can quarantine and purchase there from, each cwd said infected farm, but how many states can afford this for all the cwd infected cervid game ranch type farms ???
Tuesday, December 20, 2011
CHRONIC WASTING DISEASE CWD WISCONSIN Almond Deer (Buckhorn Flats) Farm Update DECEMBER 2011
The CWD infection rate was nearly 80%, the highest ever in a North American captive herd.
RECOMMENDATION: That the Board approve the purchase of 80 acres of land for $465,000 for the Statewide Wildlife Habitat Program in Portage County and approve the restrictions on public use of the site.
SUMMARY:
http://dnr.wi.gov/about/nrb/2011/december/12-11-2b2.pdf
http://chronic-wasting-disease.blogspot.com/2011/12/chronic-wasting-disease-cwd-wisconsin.html
SEE MORE USAHA REPORTS HERE, 2012 NOT PUBLISHED YET...TSS
http://www.usaha.org/Portals/6/Proceedings/USAHAProceedings-2010-114th.pdf
http://www.usaha.org/Portals/6/Proceedings/2009_USAHA_Proceedings.pdf
http://portals5.gomembers.com/portals/6/proceedings/2008_usaha_proceedings.pdf
Tuesday, April 16, 2013
Cervid Industry Unites To Set Direction for CWD Reform and seem to ignore their ignorance and denial in their role in spreading Chronic Wasting Disease
http://chronic-wasting-disease.blogspot.com/2013/04/cervid-industry-unites-to-set-direction.html
please see what the U.K. DEFRA recently said ABOUT CWD RISK FACTORS ;
Friday, December 14, 2012
DEFRA U.K. What is the risk of Chronic Wasting Disease CWD being introduced into Great Britain? A Qualitative Risk Assessment October 2012
snip...
In the USA, under the Food and Drug Administration’s BSE Feed Regulation (21 CFR 589.2000) most material (exceptions include milk, tallow, and gelatin) from deer and elk is prohibited for use in feed for ruminant animals. With regards to feed for non-ruminant animals, under FDA law, CWD positive deer may not be used for any animal feed or feed ingredients. For elk and deer considered at high risk for CWD, the FDA recommends that these animals do not enter the animal feed system. However, this recommendation is guidance and not a requirement by law.
Animals considered at high risk for CWD include:
1) animals from areas declared to be endemic for CWD and/or to be CWD eradication zones and
2) deer and elk that at some time during the 60-month period prior to slaughter were in a captive herd that contained a CWD-positive animal.
Therefore, in the USA, materials from cervids other than CWD positive animals may be used in animal feed and feed ingredients for non-ruminants.
The amount of animal PAP that is of deer and/or elk origin imported from the USA to GB can not be determined, however, as it is not specified in TRACES. It may constitute a small percentage of the 8412 kilos of non-fish origin processed animal proteins that were imported from US into GB in 2011.
Overall, therefore, it is considered there is a __greater than negligible risk___ that (nonruminant) animal feed and pet food containing deer and/or elk protein is imported into GB.
There is uncertainty associated with this estimate given the lack of data on the amount of deer and/or elk protein possibly being imported in these products.
snip...
36% in 2007 (Almberg et al., 2011). In such areas, population declines of deer of up to 30 to 50% have been observed (Almberg et al., 2011). In areas of Colorado, the prevalence can be as high as 30% (EFSA, 2011).
The clinical signs of CWD in affected adults are weight loss and behavioural changes that can span weeks or months (Williams, 2005). In addition, signs might include excessive salivation, behavioural alterations including a fixed stare and changes in interaction with other animals in the herd, and an altered stance (Williams, 2005). These signs are indistinguishable from cervids experimentally infected with bovine spongiform encephalopathy (BSE).
Given this, if CWD was to be introduced into countries with BSE such as GB, for example, infected deer populations would need to be tested to differentiate if they were infected with CWD or BSE to minimise the risk of BSE entering the human food-chain via affected venison.
snip...
The rate of transmission of CWD has been reported to be as high as 30% and can approach 100% among captive animals in endemic areas (Safar et al., 2008).
snip...
In summary, in endemic areas, there is a medium probability that the soil and surrounding environment is contaminated with CWD prions and in a bioavailable form. In rural areas where CWD has not been reported and deer are present, there is a greater than negligible risk the soil is contaminated with CWD prion.
snip...
In summary, given the volume of tourists, hunters and servicemen moving between GB and North America, the probability of at least one person travelling to/from a CWD affected area and, in doing so, contaminating their clothing, footwear and/or equipment prior to arriving in GB is greater than negligible. For deer hunters, specifically, the risk is likely to be greater given the increased contact with deer and their environment. However, there is significant uncertainty associated with these estimates.
snip...
Therefore, it is considered that farmed and park deer may have a higher probability of exposure to CWD transferred to the environment than wild deer given the restricted habitat range and higher frequency of contact with tourists and returning GB residents.
snip... see full text report here ;
Friday, December 14, 2012
DEFRA U.K. What is the risk of Chronic Wasting Disease CWD being introduced into Great Britain? A Qualitative Risk Assessment October 2012
http://chronic-wasting-disease.blogspot.com/2012/12/defra-uk-what-is-risk-of-chronic.html
snip...see my full submission to the state of Missouri on CWD here ;
Sunday, June 09, 2013
Missouri House forms 13-member Interim Committee on the Cause and Spread of Chronic Wasting Disease CWD
http://chronic-wasting-disease.blogspot.com/2013/06/missouri-house-forms-13-member-interim.html
continued in part 2...kind regards, terry